A new molecular link between inflammation and cancer, discovered through experiments with mice, has revealed how the body's natural repair response to tissue injury can actually spur tumor growth.
最近的一项利用小鼠进行的实验表明,人体对于组织损伤产生的自然的修复反应确实可以刺激肿瘤的生长,而这也为炎症反应与癌症之间提示了一个新的分子生物学上的关联。
RAS inhibition can reduce cell apoptosis of alveolar epithelial cells and endothelial cells, interfere with the inflammation cascade, and decrease fibroblast activity during tissue repair process.
RAS抑制可以通过多种分子机制抑制肺泡上皮细胞和血管内皮细胞的凋亡,抑制炎症级联反应和细胞外基质沉积,而使肺纤维化病变减轻。
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